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However, the therapeutic utility of doxorubicin is undermined by its dose-dependent cardiotoxicity, possibly arising as a result of futile redox cycling, with NADH dehydrogenase within the mitochondria proposed as the most likely site of anthracycline reduction 9,10

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Peer review Peer review information Nature thanks Seung-Kyun Kang, who co-reviewed with Jae-Young Bae, and the other anonymous reviewer for their contribution to the peer review of this work

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