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The authors confirmed that AngIV analogs can inhibit HGF binding to c-Met and HGF-dependent signaling by interfering with HGF dimerization, a crucial step for c-Met activation
10.1016/j.freeradbiomed.2008.04.007 15 BhadauriaM
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Acta Neurol Scand 1985, 72: 129-139
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