Presented for research literature review only Zawilska J et al
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The research team then calculated the wet/dry ratio of each specimen in order to compare them to the values of the control groups
Conclusion This study demonstrates that atorvastatin (ATO) disrupts hepatic redox homeostasis and antioxidant defenses, leading to mitochondrial dysfunction, activation of apoptotic pathways, and functional impairment of hepatic CYP3A1 activity, with associated hepatic alterations