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Food and Drug Administration (FDA) for the treatment, prevention, or cure of any disease or medical condition

Considering the positive impact of Nrf2 in ameliorating oxidative stress, a number of bioactive components that exert protective effect against APAP-associated hepatotoxicity have been identified and reported, including caffeic acid that activates the Keap1-Nrf2 antioxidant defense system (Pang et al., 2016), carnosic acid that promotes nuclear translocation of Nrf2 (Guo et al., 2016), and esculentoside A that activates Nrf2 via AMP-activated kinase/Akt/glycogen synthase kinase 3 pathway (Wang et al., 2016)

Detoxify the body by removing harmful toxins and free radicals

Dosing protocols are not standardized

GHK-Cu's mechanism centres on Cu(II) carrier function and broad gene-regulatory effects