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found through experiments that compared to the TMAO group, synaptic structural damage significantly improved in the AOB group, characterized by regular synaptic morphology, enhanced vesicle distribution within the presynaptic region, and more obvious synaptic clefts

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These represent the cutting edge of peptide-based autoimmune research, and while most have not yet reached widespread clinical application, their mechanisms and preliminary results warrant serious attention from researchers in this field

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