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Studies using rat incisional wound models demonstrated that BPC-157 treatment significantly increased tensile strength of healing tissue and improved collagen organization within the wound

Preclinical investigations demonstrated that ERR activation could influence metabolic gene expression, mitochondrial activity, and oxidative energy pathways

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It activates the same fat-cell receptor pathway (beta-3 adrenergic) as AOD 9604, triggering stored fat to be released and burned