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In Graves disease, the hyperthyroid state results from an autoimmune reaction in which antibodies overstimulate the follicle cells of the thyroid gland
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[10] It is proposed that GHK-Cu modulates copper intake into cells
This self-perpetuating loop drains the cells glutathione reserves, as the antioxidant is rapidly consumed in a desperate attempt to neutralize the surging ROS
The solution is simple: start lower than you think necessary and increase gradually
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