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They decided then to use this system to study the effects of pathologically altered red blood cells associated with sickle cell disease (SCD) and malaria, with or without associated inflammatory mediators, in the endothelial barrier function

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The most studied mechanisms include: upregulation of growth hormone receptors in fibroblasts (increasing sensitivity to healing signals), activation of the FAK-paxillin pathway (promoting cell adhesion and migration), modulation of the nitric oxide system through Src-Caveolin-1-eNOS phosphorylation (enhancing vascular function), and dual activation of the Egr-1/NAB2 gene loop (promoting organized, non-chaotic blood vessel growth)

J Am Soc Nephrol