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Administration of spironolactone, a selective MR antagonist, counteracted stress-induced mGlu2 suppression and the related development of depressive-like behavior, implicating rapid action of glucocorticoids via a receptor that is known to modulate glutamate release [20]

Vitamin-D-binding protein contributes to the maintenance of alpha cell function and glucagon secretion

This mechanistic difference is central to why the two are studied as a pair

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