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Hepatic PGC-1 overexpression exacerbates APAP-induced hepatotoxicity and mortality in mice Since Pgc-1 is one of the master regulators of liver metabolism and given its increased expression observed when mice were challenged with HFD, we decide to decipher whether PGC-1 induction could contribute to the worsening of APAP-induced hepatotoxicity
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Viborg AH, Terrapon N, Lombard V, Michel G, Czjzek M, Henrissat B, et al
Ideal candidates typically include adults with type 2 diabetes who have inadequate glycaemic control (HbA1c above individualised target) despite treatment with metformin and/or other oral agents, particularly those with additional cardiovascular or renal risk factors
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