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In this study, we investigated the hypothesis that increased clearance was due to loss of glutamine synthetase (GS), a target of beta-catenin that regulates mTORC1 activation and thus autophagy
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Straub LG, Funcke JB, Joffin N, Joung C, Al-Ghadban S, Zhao S, Zhu Q, Kruglikov IL, Zhu Y, Langlais PR, et al
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