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First, the experimental design did not include a remogliflozin-only group, which limited our ability to distinguish the direct effects of remogliflozin from its interaction with TAA-induced pathology or potential vehicle effects

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The GSTK1/RETREG1 complex conferred protection against diabetic tubular damage and kidney injury via modulating ER stress and apoptosis through the induction of reticulophagy, thereby revealing that maintaining ER homeostasis is crucial for the control of apoptosis (72, 111)

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