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Nuclear PD-L1 further complexes with pSTAT3, transcriptionally upregulating IL-6 and CXCL1 and enhancing MDSC recruitment and resistance to ICBs
The pathogenesis of PRP is primarily attributed to the dysregulation of immune function of epidermal keratinocytes, with only sparse lymphocytic infiltration observed around superficial dermal blood vessels in the uppermost dermis 6,7
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