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This rapid neutralization prevents ROS from tearing electrons away from healthy cellular structures, effectively putting out the biochemical fires driving chronic inflammation
Dihexa, with its extraordinary synaptogenic potency, is theoretically ideal for the post-stroke recovery phase when the goal shifts from neuroprotection to functional reorganization
doi.org/10.1016/j.ejphar.2007.05.072 Hsieh, M
Current Medicinal Chemistry, 29(1), 104-138
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