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Discussion The present study investigated polymorphic expression in four classes of glutathione S-transferases (GSTA1, GSTM1, GSTT1 and GSTP1) in individuals with ASD, as well as their possible gene-gene and gene-environment interaction underlying this disorder

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The MD studies revealed that very few residues, particularly within the TMD, formed stable peptide contacts, instead these interactions were transient (Supplementary Fig