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As such, it is possible that redox-associated extrahepatic mechanisms may contribute to the activation of the hepatic AMPK pathway
Resistin appears to mediate the pathogenesis of atherosclerosis by promoting endothelial dysfunction, vascular smooth muscle cell proliferation, arterial inflammation, and formation of foam cells
What Is NAC (N-Acetyl Cysteine)
The aforementioned impaired ability to neutralize ROS may also increase the risk of reperfusion injury in this patient population
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