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Furthermore, this research demonstrated that CU could degrade the levels of -syn by enhancing the autophagic function of nerve cells, suggesting that CU may mitigate oxidative stress-induced PD through multiple pathways
vi Penna A, et al
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The oxidative DNA damage in the previous investigation showed a significant increase in the asthma model [52]
When deficient, it can increase the accumulation of homocysteine, which is significant in the development of cerebrovascular diseases and cognitive impairment (4, 20, 58)
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