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[8] There is a theoretical concern about stimulating angiogenesis in the setting of undiagnosed malignancy, since VEGF upregulation by GHK-Cu could theoretically support tumor vasculature
doi: 10.3389/fcell.2021.639851 168 LiuQLiuCQYiWZOuyangPWYangBFLiuQet al
It stimulates pituitary release of endogenous GH, which can lead to downstream increases in insulin-like growth factor-1, or IGF-1
These alterations were notably reversed following treatment with WMSP (Figure 4CE)
As nitric oxide signaling and antioxidant activity become enhanced, secondary inflammation reduces
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