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The authors noted that this proliferation-inhibiting effect could have potential utility in anti-tumor applications, though further studies are needed to confirm its relevance in oncology contexts [43]

While the precise role of ROS in the pathophysiology of AD has yet to be fully understood, numerous studies have shown that dietary factors which behave as antioxidants, modulate oxidative stress and gene expression of antioxidant enzymes

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Previous studies have shown that the mechanism by which chemotherapy drugs induce tumor cell death primarily involves the promotion of ROS production [14]

Conclusion: Our results indicate that activation of the PGE2 EP4 receptor effectively reduces fibrosis in both rat and human cirrhotic PCLS