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Therapeutically, this raises two complementary possibilities: (1) SQOR-high tumors may exhibit enhanced mitochondrial ferroptosis resistance through maintained CoQ 10 H 2 and sulfur-based RTAs and (2) tumors with altered sulfur/selenium metabolism may reveal synthetic vulnerabilities when SQOR-linked defense is impaired
If any part of that chain is wrong, the final amount can be wrong too
This felt more like a rash
Acknowledgements Author contributions: K
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