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Animal studies in diet-induced obesity models show tesofensine treatment produces selective reduction in adiposity with improvements in insulin sensitivity, reductions in plasma lipids, and decreased accumulation of abdominal and subcutaneous fat mass
By converting to its active forms within the body, it supports methylation and the synthesis of nucleic acids and amino acids, which are vital for maintaining healthy nerve cells and forming red blood cells, thus correcting Vitamin B12 deficiency and associated symptoms
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Instead, coverage is contingent upon a principle known as medical necessity
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