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ALS and Dystrophic Muscle Models MGF E-peptide was shown to increase progenitor cell populations in ALS (amyotrophic lateral sclerosis), dystrophic, and normal rodent muscle establishing a research application in neuromuscular disease models beyond straightforward exercise-induced muscle damage
Inclusion criteria were: (a) RCTs
Sci (New York NY) (2016) 352(6292):140812
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Due to loss of functional OCTN2, primary carnitine deficiency patients exhibit urinary carnitine wasting
DOI: 10.2147/JEP.S35396 Beck DE, Sweeney WB, McCarter MD, et al
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