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This chronic activation impairs neuronal homeostasis, worsens synaptic dysfunction, and ultimately accelerates neurodegenerative processes [113116]

Preliminary research suggests that these mechanisms may be similar to those observed with exogenous hGH therapy, including the activation of the adrenergic receptors and increased lipolysis in adipocytes [5]

This is exactly why these medications should be used as part of a comprehensive program that includes nutrition education, behavioral support, and a long-term maintenance strategy

Data were pooled using random-effects models with subgroup analyses by drug type and treatment duration (3, 6, and 12 months)

Consistent with the pharmacological results, the above results provide convincing evidence that the GPX4 overexpression protects TECs from ferroptosis and mitigates renal interstitial fibrosis