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Retrieved 2026-01-08
demonstrated that in a lipid-rich extracellular environment, high extracellular lipid availability further enhances FA flux in BC cells, and elevated palmitate levels induce lipid toxicity, at which point exogenous FA is accumulated to be converted into triglycerides and stored in LDs to protect BC cells from palmitate-induced apoptosis
Third-party testing verification becomes essential given the peptide's sensitivity to degradation under poor handling conditions
Erickson, B
MicroRNA-specific therapeutic targets and biomarkers of apoptosis following myocardial ischemiareperfusion injury
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