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Potency of these variant fusions ranged over several orders of magnitude and some of the mutations at positions G2, L26 and I23 resulted in partial agonism (Fig
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Decreased GSH concentrations were noted in patients with alcoholic (2.55 0.1 mol/g of liver) and nonalcoholic liver diseases (2.77 0.1 mol/g of liver) compared with controls (4.14 0.1 mol/g of liver) that were speculated to be a contributing factor in liver injury and could enhance the risk of hepatic toxicity from a variety of toxic agents
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