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Discussion: Our findings demonstrate that glutathione metabolism serves as a pivotal hub in the pathogenesis of SAE
This is the same mechanism that makes GLP-1 agonists like semaglutide effective for weight management
Studies have investigated its effects in models of nerve damage and neuroinflammation, with findings suggesting neuroprotective activity and potential influence on nerve growth factor (NGF) expression
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41 Further studies also demonstrated that MLCK activation triggers caveolin-1-dependent endocytosis of occludin to effect structural and functional TJ regulation
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