and seed funds from the NIH National Center for Advancing Translational Science Clinical and Translational Science Award (UL1TR001085), SPARK (a translational research programme at Stanford), Weston Havens Foundation, and the Stanford Child Health Research Institute Transdisciplinary Initiatives (TIP) grant
doi:10.1113/jphysiol.2013.255364 Tamai I, Ohashi R, Nezu J, Yabuuchi H, Oku A, Shimane M, Sai Y, Tsuji A (1998) Molecular and functional identification of sodium ion-dependent, high affinity human carnitine transporter OCTN2
GLP-1RAs, such as lixisenatide, not only rely on -cell function and glucagon suppression, but also assist to lower glucose by other (insulin-independent) mechanisms such as delayed gastric emptying, underlying the clinical utility of GLP-1RAs as adjuvant therapy to basal insulin in longstanding T2DM [14]
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