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Modulation of the permeability transition pore Studies have reported that mitochondria permeabilize due to the mitochondrial permeability transition pore (mPTP) opening, a pathophysiological event that, under favorable conditions, causes an increase in permeability of the inner mitochondrial membrane and leads to mitochondrial depolarization, swelling, and the activation of the apoptotic and necrotic pathways 43
The study concluded that the neuroprotective effect of the EDR peptide is mediated at the molecular epigenetic level through direct interaction with DNA, thereby regulating the expression of genes responsible for neuroplasticity and preventing dendritic spine loss
2018), seems to be sufficient for redox-homeostasis in this compartment and even to supply glutathione for redox-balancing of the cytosol (Maughan et al
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