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May 28, 2026
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INTRODUCTION DKD arises from a complex interplay of metabolic, inflammatory, and hemodynamic disturbances, differing notably between type 1 and type 2 diabetes mellitus [19,20]
In addition, exendin-4 treatment repressed mitochondrial dysfunction, as shown by lower levels of the mitochondrial function-associated genes MCU and UCP3, as well as the mitochondrial fission genes DRP1 and FIS1
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