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Macrophage activation and foam cell formation may explain the elevated CRP serum levels and contribute to disease progression (Figure 3)

The dialogue within the global Sermo community reveals a nuanced perspective

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308 Rather than harboring stable resistance mutations, DTP cells adopt a slow-cycling or quasiquiescent phenotype driven by extensive epigenetic remodeling, transcriptional plasticity and metabolic reprogramming, which collectively constitute core maintenance programs that stabilize the DTP state under therapeutic pressure