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reported that BPC157 promotes angiogenesis via VEGFR2 upregulation [16], how BPC157 interacts with VEGFR2, whether it physically binds, and the mechanism underlying VEGFR2 upregulation remain unclear

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European journal of nutrition, 63(8), 30373046

doi: 10.3390/biology10030184 38 ShaWHuFXiYChuYBuS

Although our cerebral GSH results do not support the redox hypothesis of autism in the brain, previous results of decreased GSH in the plasma of autistic children may help explain some other inflammatory symptoms that autistic children experience (e.g