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Patients with insufficient glutathione stores (e.g., alcoholics and acquired immunodeficiency syndrome patients) and patients with induced cytochrome P-450 enzymatic activity (e.g., alcoholics and those taking concurrent anticonvulsant or antituberculous medications) may be at greater risk for developing acetaminophen-induced hepatotoxicity following overdose (as opposed to therapeutic dosing described earlier)
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And because glutathione production and recycling is an ATP-intensive process, you need to make sure that nothing impairs the production of ATP in your cells
Common side effects include nausea, mucositis, liver toxicity, and bone marrow suppression, necessitating close monitoring during treatment
Emodin attenuates acetaminophen-induced hepatotoxicity via the cGAS-STING pathway
