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Research in rats has observed that the peptide may substantially increase the collateral blood vessel growth rate in the setting of ischemia
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The following is a summary of documented tissue systems from peer-reviewed literature: This cross-tissue activity profile is directly attributable to the fact that its primary mechanisms VEGFR2-driven angiogenesis and NO system modulation are not tissue-specific
[1] [2] This occurs because B12 deficiency commonly affects the peripheral nerves, particularly those extending to the extremities
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