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suggested that TB-500 may reduce TLR-mediated inflammatory signaling within oligodendrocyte progenitor cell cultures by elevating miR-146a, a regulatory RNA molecule that may suppress downstream IRAK1 and TRAF6 activity

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Obesity and visceral adiposity are key drivers of HFpEF development and progression, and this may be even more amplified in women, who represent the majority of people with the disease, and bear a heavier burden of symptoms and physical limitations due to HFpEF said Mikhail Kosiborod, MD, FACC, senior author of the study and a cardiologist at Saint Luke's Mid-America Heart Institute in Kansas City, Missouri

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3,4 Of note, a newer class of antidiabetic medications known as GLP-1 receptor agonists can influence both metabolic, as well as molecular pathways