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Destabilizing mutations facilitate this initial transition, and the increased rate of conversion to a pathological state likely contributes to the earlier age of onset compared with wild-type TTR amyloidosis

METs decreased when macrophages were subjected to MSC-sEVs(Fig), and when METs were co-cultured with hepatocytes, apoptosis increased and proliferation decreased, which were reversed by the inhibition of METs

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Maackiain prevents amyloid-beta-induced cellular injury via priming PKC-Nrf2 pathway