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Since PRDX2 has been shown to be glutathionylated by GSH at its cysteine residues to protect against its hyperperoxidation 41 , it is possible that elevated GSH levels in T315I-Bcr-Abl cells might prevent PRDX2 monomer formation, therefore abrogating ROS-induced replication stress
Most confusion online comes from treating any of these as if it equals "FDA approved." Only the first one does
The same can be said of many other GLP-1 medications
But a lingering question has divided the scientific community: Is the liver improving simply because the patient is losing weight, or is there something more direct at play
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