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They are formed in the body from normal metabolism (such as converting food to energy), but are increased by exposure to environmental toxins, such as cigarette smoke and air pollution

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FIGURE 2 3.3 Anti-cell proliferation and pro-apoptosis effects 3.3.1 p53-dependent cell cycle control and apoptosis p53 is a crucial gene that regulates the balance between cell proliferation and apoptosis through a variety of routes by activating its target genes p21 Waf1/Cip1 and BCL2-associated X protein (Bax)-1

Whether this dose-response relationship extends to addiction outcomes remains to be determined, but the theoretical rationale is compelling