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In FRDA, caused by a GAA trinucleotide repeat expansion in the FXN gene, adenine base editors (ABEs) could potentially treat the disease by converting A-T to G-C base pairs within the expanded repeats in the FXN gene
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Can genetic testing help me understand my response to semaglutide
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It is the same logic that pushed glutathione from oral to IV in this country a decade ago
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