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The claims related to satiety signals and lipid metabolism are best interpreted as supportive rather than curative

Drp1-dependent mitochondrial autophagy plays a protective role against pressure overload-induced mitochondrial dysfunction and heart failure

This action was seen when DPP-4 inhibitors were used both as monotherapy and as add-on to other therapies, i.e., metformin, sulfonylureas, tiazolidinediones or exogenous insulin

However, there's actually a bit of nuance to be aware of

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