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In the pituitary gland, DSIP co-localizes with numerous other signaling molecules including ACTH, MSH, TSH, and melanin-concentrating hormone

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Unfortunately, the results seen from anti-amyloid (plaque buster) clinical trials have been disappointing [9]

Studies suggest that the VEGF/VEGFR-2 pathway may be particularly relevant, as its activation seems to trigger intracellular signaling cascades that potentially influence endothelial cell migration, proliferation, and survival