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Such findings indicate that perhaps a proportion of neonatal hyperbilirubinemia secondary to G6PD deficiency has a different mechanistic pathway other than an acute hemolytic crisis with exposure to oxidants, as seen in favism

Some receive a little, but what they may be taught is not necessarily up to date or their training focused solely on PA (pernicious anaemia) and the frankly ridiculous notion that anyone without a definite diagnosis of this or the presence of anaemia does not require treatment

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Tip: use salicylic acid wash with your loofah, La Roche Posay or Cerave make good ones