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The results showed that the peptide could accelerate the healing process by the mechanism associated with accelerated granulation tissue formation, reepithelialization, dermal remodeling, and collagen deposition through ERK1/2 signaling pathway

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All use is strictly in vitro and for research purposes only

The vasoprotective effects align neatly with the mechanistic picture: a compound that promotes angiogenesis and modulates nitric oxide would predictably stabilize vascular function across a range of stressors

however, these outcomes remain confined to preclinical research and require further investigation in human studies