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Studies in mice showed that colistin toxicity is associated with the activation of (i) the mitochondrial apoptotic pathway, (ii) the death receptor pathway, and (iii) the endoplasmic reticulum pathway 20,21
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PMID: 25731775.
Among the most studied is NfL, a structural axonal protein that increases in CSF and plasma following neuronal injury
Aside from SCFAs, a recent body of research emphasizes the significance of tryptophan-derived indoles, which influence neuroimmune balance via AhR activation, resulting in tighter BBB control and less peripheral-to-central inflammatory communication [145]
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