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OxLDL components and their interaction with toll-like receptors (TLRs) 2 and 4, CD36 and other cellular receptors further mediate thromboinflammation enhancing tissue and organ damage culminating in organ failure, i.e., myocardial infarction, stroke, and pulmonary artery embolism (174, 209212)

Moreover, Fer-1 treatment significantly reduced the release of profibrotic cytokines, including TGF-, CTGF, PDGFB, and FGF2, from TECs (Fig

ROR1 also can be targeted by miR382, which might serve as another option for OC [210]

Guh DP, Zhang W, Bansback N, Amarsi Z, Birmingham CL, Anis AH

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