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Previous studies have shown that mitochondrial dysfunction related to PD can be caused by various factors, including mitochondrial bio-genetic defects, increased ROS production, mitophagy defects, transport disorders, electron transport chain dysfunction, mitochondrial dynamics changes, and mitochondrial calcium imbalance
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Even modest reductions in micronutrient availability may therefore influence catalytic velocity across interconnected metabolic pathways, potentially amplifying bioenergetic and redox constraints under chronic flux limitation [112, 113]
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