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These processes compromise neurogenesis, neuronal migration, and synapse formation (Komada and Nishimura 2022)

In principle, a hyperoxidizing ER might simply blunt the UPR rather than improve ER function, since activation of at least the UPR sensors ATF6 and IRE1 can be inhibited by oxidation (Nadanaka, Okada, Yoshida, & Mori, 2007