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Clinical research has documented that glutathione depletion impairs immune function, while repletion restores it (Drge & Breitkreutz, 2000)

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Within the signal transducer and activator of the transcription (STAT) family, STAT1 promotes the expression of pro-inflammatory genes upon activation by IFN-, whereas STAT3 attenuates inflammation by inhibiting NF-B under the influence of IL-10 signaling

While both compounds act as GHRH analogues, they differ in their structural durability