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The occlusion cases ratio was up to ~76% at 1.5 mM of tBuOOH (Supplementary Fig

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These medications work by stimulating the GLP-1 receptors in the body, which leads to the same effects as the naturally produced hormone

Moreover, mitochondrial dysfunction and related oxidative stress can activate the innate branch of the immune system, leading to the release of pro-inflammatory cytokines influencing various depression-related pathophysiological mechanisms: monoaminergic neurotransmission disruption, tryptophan degradation toward neurotoxic catabolites, glutamate-related excitotoxicity, decreased neurotrophic factors and alterations in the hypothalamic-pituitary-adrenal axis [3, 33, 59]

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