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The mechanism underlying LP in mammalian cells is not well understood

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Interestingly, previous studies suggested that increased UA may be associated with protein decomposition and forging behavior during the fasting period [51]

Biochemically, once ferroptosis is activated, it cannot be suppressed by apoptosis, necrosis, or autophagy inhibitors but can be hidden by iron chelators such as deferoxamine (DFO) or antioxidants such as Trolox and fer-1

405 This suggests that signaling through GLP-1R in astrocytes is crucial for maintaining the metabolic stability and functionality of the cells