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Their effects are fairly minor, but they may both be beneficial for improving recovery, as well as being useful medical treatments in certain cases

GLP-1 analogs have been shown to modulate neurotransmitter release related to hunger and reward in the hypothalamus and striatum, resulting in reduced food intake [13]

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The third approach aims to enhance endogenous GLP-1 production from intestinal L-cells

This adaptation period likely reflects physiological tolerance to the drug's effects on gastric emptying and gastrointestinal function